odontoblast การใช้

• The odontoblast process causes the secretion of hydroxyapatite crystals and mineralization of the matrix.
• Enamel spindles however, are in fact odontoblast processes that extend into the enamel.
• This is because they are formed by entrapment of odontoblast processes between ameloblasts prior to and during amelogenesis.
• The odontoblasts begin to move toward the center of the tooth, forming an extension called the odontoblast process.
• The differentiation of the odontoblast is done by signaling molecules and growth factors in the cells of the inner enamel epithelium.
• Dmp4 causes differentiation of mesenchymal stem cells into functional odontoblast cells and is likely to function as a regulator of dentin mineralization.
• Although NBPF10's function is unknown, there is reason to believe that NBPF10 is an important biomarker for the Odontoblast Phenotype
• The dentinal fluid in the tubule presumably also includes the tissue fluid surrounding the cell membrane of the odontoblast, which is continuous from the cell body in the pulp.
• In an erupted tooth, this process rarely extends beyond 1 / 3 the depth of the dentin, which is why the odontoblast transduction theory of dentinal hypersensivity is unlikely.
• Enamel spindles are also linear defects, but they too can be found only at the DEJ, because they are formed by entrapment of odontoblast processes between ameloblasts prior to and during amelogenesis.
• It is of two types, either reactionary, where dentin is formed from a pre-existing odontoblast, or reparative, where newly differentiated odontoblast-like cells are formed due to the death of the original odontoblasts, from a pulpal progenitor cell.
• It is of two types, either reactionary, where dentin is formed from a pre-existing odontoblast, or reparative, where newly differentiated odontoblast-like cells are formed due to the death of the original odontoblasts, from a pulpal progenitor cell.
• A pulpal A-delta ( noxious, short sharp pain ) nerve fibre is either wrapped around the base of this process, or travels a short way into the dentinal tubule with the odontoblast process ( max ~ 0.1 mm ) This process lies in the dentinal tubule.
• In the case of an infection breaching the dentin to or very near the pulp, or in the instance of odontoblast death due to other attack ( e . g . chemical or physical ), undifferentiated mesenchymal cells can differentiate into odontoblast-like cells which then secrete another type, reparative dentin, underneath the site of attack.
• In the case of an infection breaching the dentin to or very near the pulp, or in the instance of odontoblast death due to other attack ( e . g . chemical or physical ), undifferentiated mesenchymal cells can differentiate into odontoblast-like cells which then secrete another type, reparative dentin, underneath the site of attack.
• Tertiary dentin is only formed by an odontoblast directly affected by a stimulus; therefore, the architecture and structure depend on the intensity and duration of the stimulus, e . g ., if the stimulus is a carious lesion, there is extensive destruction of dentin and damage to the pulp, due to the differentiation of bacterial metabolites and toxins.
• My journey : ( 1 ) is to find out if this is related to the 3-series puppy-shot that contain " parvo, influenza, distemper & hep . " when administered with an undetected low-grade fever, ( 2 ) or, if there is a heredity-gene trait and is there a genetic test available ? ( 3 ) to answer a question swarming my brain; what could destroy the mineral presentation of " Hydroxyapatite " during the " Odontoblast period " of the " Dentinogenesis " formation?