postmitotic การใช้

• These remaining DNA damages accumulate with age in mammalian postmitotic tissues.
• TBR1 both positively and negatively regulates gene expression in postmitotic neurons.
• This prevents entrance of postmitotic cells both into MC and apoptosis.
• Each preameloblast elongate and becomes an postmitotic, polarized, secretory ameloblast.
• Studies in mice suggest that the protein encoded by this gene may suppress growth in postmitotic neurons.
• The expression of CRMPs also can be found in the death or survival signaling of postmitotic neurons.
• KIF15 co-localizes with microtubules and actin filaments in both dividing cells and in postmitotic neurons.
• Telomere shortening does not occur with age in some postmitotic tissues, such as in the rat brain.
• It was discovered that Tbr-1 is expressed by postmitotic cortical neurons in mice and in humans.
• Also, telomere shortening does not occur with age in a some postmitotic tissues, such as in the rat brain.
• During this migration, there are three stages of cellular development : radial glia, intermediate progenitors, and postmitotic projection neurons.
• Being a more fate restricted cell the radial glial cell will either generate postmitotic neurons, intermediate progenitor cells, or astrocytes in gliogenesis.
• Tbr1 is expressed mainly in early-born postmitotic neurons of the developing cerebral cortex in particular, the preplate and layer VI neurons.
• Neuronal Nuclei antigen ( NeuN ) or Fox-3 is a nuclear protein present in postmitotic stage when start to differentiate into mature cells.
• Only in postmitotic cells ( i . e . cells in early G 1 ) did serum withdrawal force cells into quiescence ( G 0 ).
• Only postmitotic cells younger than three hours arrested cellular division in these conditions, while cells older than four hours were insensitive to the absence of growth factors.
• KIF15 ( also known as Kinesin-12 and HKLP2 ) is a motor protein expressed in all cells during mitosis and in postmitotic neurons undergoing axon growth.
• Neuron-specific Class III ?-tubulin ( TuJ1 ) is present in newly generated immature postmitotic neurons and differentiated neurons and in some mitotically active neuronal precursors.
• Telomerase lengthens telomeres in DNA strands, thereby allowing senescent cells that would otherwise become postmitotic and undergo apoptosis to exceed the Hayflick limit and become potentially immortal, as is often the case with cancerous cells.
• This occurs because of its interaction with ERK ( extracellular signal-regulated kinase ) molecules involved in the regulation of meiosis, mitosis, and postmitotic functions in differentiated cells, whose pathway can be activated by carcinogens or viral infections ..